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By John R. Baker, Ralph Muller, David Rollinson

ISBN-10: 0120317583

ISBN-13: 9780120317585

The Advances in Parasitology sequence includes in-depth stories on present themes of curiosity in modern parasitology. It comprises scientific stories on parasites of significant impression, reminiscent of trypanosomiasis and scabies, and extra conventional parts, resembling zoology, taxonomy, and lifestyles historical past, which form present pondering and purposes. * sequence has the second one maximum ISI effect think about the parasitology staff! (4.818 in 2002) * participants are foreign specialists within the box

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Download e-book for iPad: Advances in Parasitology, Vol. 58 by John R. Baker, Ralph Muller, David Rollinson

The Advances in Parasitology sequence includes in-depth reports on present themes of curiosity in modern parasitology. It contains scientific experiences on parasites of significant effect, akin to trypanosomiasis and scabies, and extra conventional parts, reminiscent of zoology, taxonomy, and existence heritage, which form present considering and functions.

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Extra info for Advances in Parasitology, Vol. 58

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Braziliensis induces both splenic DC migration from marginal zones to T cell areas and splenic DC maturation. , 2004) vs. , 2002), which prevents doing strict comparisons of the results. In any case, the fact that in the study of De Trez et al. (2004), DC migration and maturation are observed only after injecting a huge number of promastigotes (108 to 5 Â 108) makes the results questionable. 2. MHC I and MHC II Ag Presentation by DCs Put in Contact with Parasites or Leishmania Ags Accurate studies of the sensu stricto Ag presentation processes occurring in Leishmania-infected DCs are still scarce.

C. ANTOINE ET AL. , 1998; Overath and Aebischer, 1999). , 1999). These data are consistent with the idea that only MÈs containing parasites that are rapidly killed after internalization are able to present some Leishmania Ags, including Ags expressed at the parasite plasma membrane. However, parasite killing is not always sufficient for Ag presentation by infected MÈs. , 1999). Thus, parameters other than the resistance to killing must be involved in the weak presentation or in the lack of presentation of parasite Ags characterizing MÈs initially infected with metacyclics or amastigotes.

31 activation/reactivation of parasite-specific T cells. Several studies using mouse or dog MÈs as host cells have demonstrated that L. donovani or L. , 1999). , 1994). These findings suggest that infected MÈs are probably not involved in the activation of naive Leishmania-specific T cells, in line with the fact that L. , 1999) or are endowed with a poor priming potential compared to DCs (Shankar and Titus, 1997). 5. In vivo Data Most of the conclusions concerning the Ag presentation capacity of Leishmania-infected MÈs were drawn from in vitro experiments and their physiological relevance can thus be questioned.

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Advances in Parasitology, Vol. 58 by John R. Baker, Ralph Muller, David Rollinson


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